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大学英语四级考试阅读理解强化训练(新题型)

作者:丛力,
分类:文学
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装帧:平装(无盘) / 260×185×5毫米 / 168页 / 0字
ISBN(10位/13位):781046471X
出版:上海外语教育出版社2000-03- 1出版
定价:¥12元

标签(Tags):外语类考试  
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Passage 1

For as long as biologists have studied cancer,they have held one dream.Their hope has been that,for all of the many different cancers that can eat away at a body there will turn out to be a single step that determines whether cells turn cancerous. By targeting at that step,physicians would be able to stop a cell from heading down the path toward cancer just as a switchman stops a train from going down the wrong track. It turns out that a gene(基因)called p53may be that switch. If p53 gene is healthy and doing its job,it keeps the cell on the track of normality. But if the p53is absent,damaged or tied up by other molecules,the cell goes down the track toward cancer. "p53 fulfills this long-held hope,"says Dr.Bert Vogelstein of the Howard Hughes Medical Institute(HHMI). "Alterations of the p53 gene appear to be involved,directly or indirectly,in the majority of human cancers." All told,p53 has been implicated in more kinds of cancer than most people knew existed.

Not bad for a gene that first broke on the scene in a bad case of mistaken identity(特性). In 1979,David Lane of the University of Dundee in Scotland and Arnold Levine of Princeton University independently discovered p53,the protein that the p53 gene makes. In 1982 biologists isolated the p53 gene. But it seemed to cause,rather than suppress(抑制),cancer. Few researchers were interested in yet another one of those. It was not until 1989 that Levine and Vogelstein separately discovered p53s true colors:it was a tumor killer.With this realization,some 2,000 biologists have now dropped the date they brought to the dance — their previous research subject — and became interested in the pretty new arrival.
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